I've been thinking of writing a series on viral parkinsonism after your case report on the septuagenarian with pre-diagnosed Parkinson's being exacerbated by the vaccines but shelved that. It appears you were able to get to many of those points! Ironically, I'm also looking a bit at the microbiome in my recent posts, so maybe hold off before you dive deeper into that first! 😉
There's a lot going on right now that seems very scary, but under many circumstances the fear can override rational thought. If context is removed then it's easy to see things from only one perspective.
Many things would need to be looked deeper, and we do need to find answers but we also require a lot of context when examining information.
or the prion insert? Prions disease anyone? I have personally witnessed my children have seizures, alice in wonderland syndrome from whatever respiratory frankengarbage is floating around thanks to mans folly several times since the plandemic began, and believe me its terrifying and I do not believe for one minute benign.
Given that plant viruses can induce both growth and restriction in plant cells, leading to all manner of tissue differentiation ("disturbances") one has to wonder if plant virus genes should be thought of any differently from endogenous plant genes that regulate differentiation. There's no reason why plant genes wouldn't come from viruses and vice-versa.
Likewise for insects (metamorphosis) and animals (embryogenesis). If papilloma viruses can direct cells to divide, then they might be related to ancient versions of the same genes that we use to do the same. All of our genes could be a symphony of ancient viruses.
Yes, I saw - I consider LBC footnotes a must-read before commenting.
Given that the most famous example is ERVW1 / syncytin, one wonders whether fusion protein-bearing retroviruses were essential in distributing evolutionary advances throughout multiple mammalian branches (as opposed to all placental mammals having a common ancestor or achieving convergent evolution).
For insects, symbiotic intracellular bacteria / endosymbionts are to them what plasmids are to bacteria. Whereas vertebrates, besides mitochondria, don't seem to "work well with others" as much when it comes to other potential intracellular bacterial partners (like spirochete).
Actually that makes me wonder whether there's something about having an adaptive immune system like what vertebrates have, which makes it so that we don't work well with others in the way that invertebrates or plants do... That would be an interesting rabbit hole to go down
Good question! They were talking about locating viral RNA in the brains, NOT about looking for the deadly spike proteins! (From the shots, obviously, if they were present but there were no accompanying viral envelope proteins.)
Couple of points- the lack of detection of SarsCov2 in the brain, despite apparent infection/distribution elsewhere, the body will protect the organs most required for life, even to the perceived detriment on other areas. Also anything that affect CNS or specifically vagus nerve (which SarsCov2 does) generally exhibit "neurological symptoms". Be it as simple as lowered GABA and subsequent side effects in response.
There are differences in distribution data between wild type SarsCov2 infection and "SarsCov2 vaccine" distribution.
Finally, your discussion and suggestions on plant biogy are spot on. In fact so much so, that when you look at certain microbes within the soils that have been systematically destroyed from conventional and even organic agriculture practices, we find that it affects the animals that consume vegetable matter grown in these depleted soils (Or the secondary consumption of animals consuming this matter), so much so that specific brain chemicals are dysregulated or even not produced at all. Which when combined with inside lifestyles and the purposeful layering of sunblock, lowers the photonic electrical gradient within cells and lowers the bodies ability to keep even commensal or non-pathogenic microbes in check, or the inflammation caused by the bodies own cellular mechanisims. Food for thought....perhaps the presence of SarsCov2 would have never even made a blip, if the worlds populations (particularly westernised countries), hadnt already had compromised eco and microbiome systems.🤔🤔😐🤦♀️🤦♀️🤯
What is less well known is the fact that we have an intrinsic relationship with viruses just as we do with the bacteria and fungi, etc., of our microbiome. Our ever-present virus population is called the "virome". Here is an article that discusses it:
The human virome: assembly, composition and host interactions | Nature Reviews Microbiology
I find something very interesting about all of this. It seems quite astonishing to me to make the statement "well it may just be dead viruses", when they observe residues of genetic material that are related to viral infections, with no pathophysiological activity within the tissues where they are found, and in sufficient quantities. As you pointed out from one of the papers "we found few histopathologic changes, despite substantial viral burden".
What this reveals, from my point of view, is that we do not understand the dynamics of those biological structures within the human ecosystem, or within the ecosystem in general. That is, we have a very deep gap in knowledge in relation to etiology, pathophysiology and omics (where I would include virology).
We seem to just jump into conclusions based on narratives, prejudice and superstition.
I would love to see studies of all the "died suddenly" bodies and what they have in common, and what might have been a causal factor. Is there a list of these? Please provide.
The media always wants to blame deaths on COVID and maintain that the shots are "safe and effective" (!). Dr. McCullough states that very few cases of myocarditis have occurred as a result of natural SARS-CoV-2 infections. (Although many circulating viruses will on rare occasions result in myocarditis.) Virtually all the cases of myocarditis and pericarditis seen in this "plandemic" are caused by the massive production of viral spikes by the COVID shots.
Myocarditis kills heart muscle cells and leaves behind scar tissue. It is that scar tissue that can disrupt electrical conductivity within the heart and trigger fatal ventriclar fibrillation: "sudden cardiac death".
Similarly, deaths that occur during sleep probably are triggered by the early morning natural increase in catecholamine "stress" hormones that elevate to wake us up in the morning. They cause an increase in heart rate and blood pressure to get us "going", but can be "the straw the broke the camel's back" with a badly damaged and scarred heart. The stressed heart goes into fibrillation and it's immediate "lights out"!
Thank you for the details. I already know that the mRNA injections are causing a lot of death, but I just wondered if Joomi might be privy to some studies that are buried from public view. I know, however, that for the most part these sudden deaths are deliberately not being investigated because there is a silent understanding in the Big Pharma-funded scientific community of what is causing them.
Couple of thoughts, are we sure every injection contains the same material, and is it possible that through the decades people have been experimenting on the populace using common cold virus?
Darn, Joomi!
I've been thinking of writing a series on viral parkinsonism after your case report on the septuagenarian with pre-diagnosed Parkinson's being exacerbated by the vaccines but shelved that. It appears you were able to get to many of those points! Ironically, I'm also looking a bit at the microbiome in my recent posts, so maybe hold off before you dive deeper into that first! 😉
There's a lot going on right now that seems very scary, but under many circumstances the fear can override rational thought. If context is removed then it's easy to see things from only one perspective.
Many things would need to be looked deeper, and we do need to find answers but we also require a lot of context when examining information.
Looking forward to reading it!
Never discount neurotoxicity from the GP120 inserts in S1, that easily crosses the blood brain barrier and doesn't require live virus.
It can persist in the axon for decades and inhibit neuron function without killing it.
I will update this review in the next couple of days with a contents and new research:
Pathophysiology of "brain fog"
https://doorlesscarp953.substack.com/p/pathophysiology-of-spike-protein
Looking forward to it
I have now finished updating with a contents page and effects of gp120 on fast axonal transport:
Pathophysiology of "brain fog"
https://doorlesscarp953.substack.com/p/pathophysiology-of-spike-protein
or the prion insert? Prions disease anyone? I have personally witnessed my children have seizures, alice in wonderland syndrome from whatever respiratory frankengarbage is floating around thanks to mans folly several times since the plandemic began, and believe me its terrifying and I do not believe for one minute benign.
Given that plant viruses can induce both growth and restriction in plant cells, leading to all manner of tissue differentiation ("disturbances") one has to wonder if plant virus genes should be thought of any differently from endogenous plant genes that regulate differentiation. There's no reason why plant genes wouldn't come from viruses and vice-versa.
Likewise for insects (metamorphosis) and animals (embryogenesis). If papilloma viruses can direct cells to divide, then they might be related to ancient versions of the same genes that we use to do the same. All of our genes could be a symphony of ancient viruses.
At the least some of our genes came from viruses and other microbes (I discussed a bit of that in the last footnote). It's a fascinating topic
Yes, I saw - I consider LBC footnotes a must-read before commenting.
Given that the most famous example is ERVW1 / syncytin, one wonders whether fusion protein-bearing retroviruses were essential in distributing evolutionary advances throughout multiple mammalian branches (as opposed to all placental mammals having a common ancestor or achieving convergent evolution).
For insects, symbiotic intracellular bacteria / endosymbionts are to them what plasmids are to bacteria. Whereas vertebrates, besides mitochondria, don't seem to "work well with others" as much when it comes to other potential intracellular bacterial partners (like spirochete).
Actually that makes me wonder whether there's something about having an adaptive immune system like what vertebrates have, which makes it so that we don't work well with others in the way that invertebrates or plants do... That would be an interesting rabbit hole to go down
How many had taken the covid death shots?
That was my question as well
Good question! They were talking about locating viral RNA in the brains, NOT about looking for the deadly spike proteins! (From the shots, obviously, if they were present but there were no accompanying viral envelope proteins.)
Thanks for giving this context! 👍🏽💕
Yes!!!!👏👏👏💯
Couple of points- the lack of detection of SarsCov2 in the brain, despite apparent infection/distribution elsewhere, the body will protect the organs most required for life, even to the perceived detriment on other areas. Also anything that affect CNS or specifically vagus nerve (which SarsCov2 does) generally exhibit "neurological symptoms". Be it as simple as lowered GABA and subsequent side effects in response.
There are differences in distribution data between wild type SarsCov2 infection and "SarsCov2 vaccine" distribution.
Finally, your discussion and suggestions on plant biogy are spot on. In fact so much so, that when you look at certain microbes within the soils that have been systematically destroyed from conventional and even organic agriculture practices, we find that it affects the animals that consume vegetable matter grown in these depleted soils (Or the secondary consumption of animals consuming this matter), so much so that specific brain chemicals are dysregulated or even not produced at all. Which when combined with inside lifestyles and the purposeful layering of sunblock, lowers the photonic electrical gradient within cells and lowers the bodies ability to keep even commensal or non-pathogenic microbes in check, or the inflammation caused by the bodies own cellular mechanisims. Food for thought....perhaps the presence of SarsCov2 would have never even made a blip, if the worlds populations (particularly westernised countries), hadnt already had compromised eco and microbiome systems.🤔🤔😐🤦♀️🤦♀️🤯
What is less well known is the fact that we have an intrinsic relationship with viruses just as we do with the bacteria and fungi, etc., of our microbiome. Our ever-present virus population is called the "virome". Here is an article that discusses it:
The human virome: assembly, composition and host interactions | Nature Reviews Microbiology
https://www.nature.com/articles/s41579-021-00536-5
Hi Joomi,
I find something very interesting about all of this. It seems quite astonishing to me to make the statement "well it may just be dead viruses", when they observe residues of genetic material that are related to viral infections, with no pathophysiological activity within the tissues where they are found, and in sufficient quantities. As you pointed out from one of the papers "we found few histopathologic changes, despite substantial viral burden".
What this reveals, from my point of view, is that we do not understand the dynamics of those biological structures within the human ecosystem, or within the ecosystem in general. That is, we have a very deep gap in knowledge in relation to etiology, pathophysiology and omics (where I would include virology).
We seem to just jump into conclusions based on narratives, prejudice and superstition.
I would love to see studies of all the "died suddenly" bodies and what they have in common, and what might have been a causal factor. Is there a list of these? Please provide.
The vast majority of those who died suddenly are never tracked or autopsied so we will never know . . . .
@DC ~ Here's a related update from Dr. Peter McCullough on a study he did of heart attacks on the playing field in European sports leagues:
> https://workflowy.com/s/beyond-covid-19/SoQPdY75WJteLUYx#/v_8b7d39587184_392d9fdd648e_6f81d0505b6b579fd2386737622f
The data is telling . . . .
The media always wants to blame deaths on COVID and maintain that the shots are "safe and effective" (!). Dr. McCullough states that very few cases of myocarditis have occurred as a result of natural SARS-CoV-2 infections. (Although many circulating viruses will on rare occasions result in myocarditis.) Virtually all the cases of myocarditis and pericarditis seen in this "plandemic" are caused by the massive production of viral spikes by the COVID shots.
Myocarditis kills heart muscle cells and leaves behind scar tissue. It is that scar tissue that can disrupt electrical conductivity within the heart and trigger fatal ventriclar fibrillation: "sudden cardiac death".
Similarly, deaths that occur during sleep probably are triggered by the early morning natural increase in catecholamine "stress" hormones that elevate to wake us up in the morning. They cause an increase in heart rate and blood pressure to get us "going", but can be "the straw the broke the camel's back" with a badly damaged and scarred heart. The stressed heart goes into fibrillation and it's immediate "lights out"!
Thank you for the details. I already know that the mRNA injections are causing a lot of death, but I just wondered if Joomi might be privy to some studies that are buried from public view. I know, however, that for the most part these sudden deaths are deliberately not being investigated because there is a silent understanding in the Big Pharma-funded scientific community of what is causing them.
They keep saying they found a virus when they just found RNA...
Couple of thoughts, are we sure every injection contains the same material, and is it possible that through the decades people have been experimenting on the populace using common cold virus?
Now, if I could make that plant grow on my fur, to give me better sun protection, that would be next level.